pathological hypertrophy
英 [ˌpæθəˈlɒdʒɪkl haɪˈpɜːtrəfi]
美 [ˌpæθəˈlɑːdʒɪkl haɪˈpɜːrtrəfi]
网络 病理性肥大
双语例句
- Objective To create the exercise-induced physiological and DOCA-salt-induced pathological cardiac hypertrophy animal model, and evaluate cardiac function during development of hypertrophy.
目的通过建立运动引起的生理性心脏肥大和DOCA高盐引起的病理性心脏肥大大鼠模型，比较评价两种心脏肥大时心脏功能情况。 - Review on comparative study between exercise-induced and pathological myocardial hypertrophy;
运动性与病理性心肌肥厚对比研究进展（综述） - Pathological cardiac hypertrophy is one of adaptive responses of the heart to a variety of pathological stimuli, and always one of the complications of some cardiovascular diseases, such as hypertension, myocardial infarction, congenital heart diseases, cardiovalvular diseases, and etc.
病理性心肌肥厚是高血压、心脏瓣膜病、急性心肌梗塞及先天性心脏病等临床常见疾病的一种并发症，是心脏对多种病理刺激的一种适应性反应。 - In ( adult,) TGF-β s are required for maintaining the normal physiological function of cardiovascular system. Alteration in TGF-β signaling is highly related to multiple heart diseases, including the pathological processes of cardiac hypertrophy, cardiac fibrosis and heart failure.
成年后，TGF-β家族分子在维持心血管系统正常生理功能方面起到重要作用，TGF-β信号通路分子的改变与心肌肥大及其向心衰的改变、心肌纤维化等病理过程密切相关。 - Conclusions: Sedimentation of protein in collagen matrix network is one of the pathological changes in hypertensive cardiac hypertrophy.
结论：高血压性心肌肥大间质胶原骨架网络区的病变是主要损伤之一； - A Comparative Study on Myocardial Mechanics between Exercise-induced and Pathological Left Ventricular Hypertrophy in Rats
运动性与病理性左室肥厚心肌力学的对比研究 - Twenty rats with exercise and pressure_overload induced myocardial hypertrophy were taken as objects, the parameters of myocardial cells, specific surface and specific membrane of mitochondria were measured to ulteriorly find out the morphological differences between endurance training induced and pathological myocardial hypertrophy.
为了解耐力运动性与病理性心脏肥大在形态学上的差异，本文在大鼠游泳训练心肌肥大和肾血管性高血压肥大模型上，测量了各组心肌细胞参数及心肌线粒体比表面与内膜比膜面。 - Diabetic cardiomyopathy is an independent diabetic complication. The main pathological manifestations are hypertrophy of cardiac muscle and fibrosis of tissue space.
糖尿病心肌病是糖尿病的独立并发症，其主要病理改变为心肌肥大和间质纤维化。 - The pathological observation and clinical significance of posterior longitudinal ligament hypertrophy in cervical spondylotic myelopathy
脊髓型颈椎病肥厚后纵韧带的病理学观察及临床意义 - Furthermore, pathological changes, such as proliferation and hypertrophy of secretion cells which potentially facilitated to mucins hypersecretion and more terminal sialic acids, exist in some chronic airway inflammatory diseases like COPD.
而COPD等气道慢性炎症还存在着分泌细胞增生肥大等可能易于发生高分泌及高度唾液酸化的病理学基础。 - Besides cardiomyocytes hypertrophy, the pathological basis of left ventricular hypertrophy shows cardiac fibroblasts proliferation and excessive extracellular matrix protein accumulation, which leads to myocardial fibrosis.
左心室肥厚的细胞病理学基础不仅表现在心肌实质细胞的肥大，还有心脏成纤维细胞（CFs）的过度增殖和细胞外基质大量沉积，导致心肌纤维化。 - Myocardial hypertrophy is an important pathological character of congestive heart failure. Myocardial hypertrophy badly harms the patients 'health and lowers their life quality.
心肌肥大是慢性心力衰竭的重要病理特征，严重危害患者健康、降低生活质量。 - Results: The pathological changes were in sequence: joint space narrowing, osteoporosis or sclerosis of the subchondral bone, osteophyte formation, vacuum joint, joint capsule calcification, sagittal articular orientation change, hypertrophy of articular processes, partial dislocation.
结果：小关节退变病理变化依次为：关节间隙狭窄、软骨下骨疏松或硬化、骨赘形成、关节真空、关节囊钙化、关节面矢向变化、关节突肥大、小关节半脱位。 - The pathological basis of left ventricular hypertrophy include myocytes hypertrophy and cardiac fibroblast ( CFs) proliferation and excessive collagen synthesis in CFs, which leads to extracellular interstitial accumlation of collagen, and affect the progression of LVH.
心肌纤维化是高血压发病过程中左室重建的重要病理改变，心脏成纤维细胞（CFs）的增殖和其生物活性决定着心肌细胞外间质胶原的生成和降解，而最终影响心肌肥厚的发生与发展。 - The classical neurotransmitters and nerve peptides play an important role not only on cardiovascular physiological regulation, but also the pathological process of myocardial hypertrophy and heart failure.
各种神经递质和肽类物质不仅对心血管系统具有重要的生理调节作用，而且在心肌肥厚、心力衰竭等病理过程中也具有重要的作用。 - The multiplication and secretion of myocardial fibrocyte are important factors influencing the structure of myocardial tissue, exercise induced myocardial hypertrophy is different from pathological myocardial hypertrophy in the structure of myocardial tissue.
心肌成纤维细胞的增殖及其旁（自）分泌功能可能是决定肥厚心肌架构的重要因素，从而揭示运动性心肌肥厚与高血压性心肌肥厚在心脏机能上的差异可能与心肌架构密切相关。 - The main pathological changes of DCM are microvascular endothelial cells impairment, collagen fibers in endocardium proliferation, vascular basement membrane thickening, myocardial interstitial inflammation and fibrosis as well as cardiomyocytes hypertrophy and apoptosis.
糖尿病心肌病的主要病理改变是心肌微血管内皮细胞损害，内膜下纤维增生，血管基底膜增厚，心肌间质炎症和纤维化，心肌细胞肥大、凋亡。 - The early pathological features of DN are glomerular hypertrophy, thickening basement membrane and accumulating Extracellular matrix ( ECM) at glomerular mesangial. The late pathological features of DN are glomerulosclerosis and interstitial fibrosis.
DN早期病理特征是肾小球肥大、基底膜增厚和肾小球系膜区细胞外基质（Extracellularmatrix，ECM）堆积，晚期则表现为肾小球硬化和间质纤维化。 - Although pathological hypertrophy of the myocardium is temporarily compensatory, prolonged hypertrophy may result in ventricular arrhythmias, heart failure, and subsequent cardiovascular mortality.
虽然心肌细胞最初的生理性肥大是一种代偿机制，但是持续长久的心肌肥厚可能导致心律失常、心力衰竭甚至猝死。 - MiR-21 may be involved in the pathological processes of hypertensive cardiac hypertrophy and renal fibrosis.
miR-21可能参与了高血压致心肌肥厚及肾脏纤维化的病理过程。 - The pathological process of myocardial ischemia, cardiac hypertrophy and heart failure include changes in the cytoskeleton.
细胞骨架变化往往会引起心肌缺氧、心肌肥厚及心力衰竭等病理性进程。